Training & Nutrition - any nutrition recommendation for bursitus?

Ive been told I have bursitus in both my shoulders the past two years and now in my hip.

Overuse of the joints is one reason but I wonder why Im prone to this and wonder if I could add anything in my diet to prevent me from getting it in another joint. I do not have a good diet, that could also be the reason Im prone to this more then others. Im ready to change this.

any ideas?

I've been reading up on inflammatory conditions recently, as I have a freind with recently diagnosed bursitis. There is some evidence that fish oil supplementation may help reduce inflammation.
We're talking 3+ grams of EPA/DHA per day. This is a lot, and can be sort of expensive, but it has fewer side effects than aspirin in most people. If you try this, usually fish oil capsules contian 1 gram of fish oil but only .3 grams of EPA and DHA combined meaning you have to take 3 pills to get 1 gram of the active omega 3's. Some pills have more concentrated EPA/DHA so check the label. Talk it over with your Dr. before taking this amount as it may effect any other meds you might be taking.

thanks! I'll check on it and with doc as well...
Im taking so much advils as of recently, I have to try something else soon!

BDG, my girlfriend's had bursitis in her shoulder - cortizone shots wound up really helping, a lot healthier than taking tons of painkillers. Ask your doctor if they're right for you. She also has a PhD in Nutritional Science, and definitely knows first hand that diet is a key factor in rebuilding everything from muscle to bone - ask your doctor if a nutritional consult might be a good idea if you think you eat that poorly.

A fellow rider also swears by fish oil, but that's (obviously) totally anecdotal - works for him, claims it's effectively joint lubrication. Might be worth a try, depends on what your risk tolerance for supplements are. I've not attempted it yet, opting instead for stretching, rest days, and improving bike fit thus far...as I get older my option may need to be expanded...

Atomick, like your gf I did get a cortisone shot in my shoulder and in my hip, it helps somewhat, I just have to manage the pain and rest when it acts up.
I guess i was wondering if I can take anything out of my diet or add new items to try and prevent this from happening to another joint.

maybe a nutritionist would be beneficial for me.

How about this:

ORIGINAL RESEARCH COMMUNICATIONS
Fish oil supplementation inhibits the expression of major histocompatibility complex class II molecules and adhesion molecules on human monocytes

DA Hughes, AC Pinder, Z Piper, IT Johnson and EK Lund
Department of Nutrition, Diet and Health, Institute of Food Research, Norwich Laboratory, Norwich Research Park, Colney, Norfolk, United Kingdom.

To test the hypothesis that fish oil supplementation can inhibit the expression of functionally associated molecules on the surface of human blood monocytes, we randomly assigned 12 healthy adults to receive either an n-3 polyunsaturated fatty acid-rich fish oil supplement for 21 d or to receive no supplement. The percentage of monocytes expressing major histocompatibility complex (MHC) class II molecules (HLA-DR, -DP, and -DQ), intercellular adhesion molecule-1, and leukocyte-function-associated antigen-1, and the intensity of expression of each molecule were quantified before and after the study period. Monocytes were examined immediately after blood sampling and again after incubation in serum-free culture medium for 24 h in the presence of interferon-gamma to up-regulate expression of MHC class II molecules by the monocytes. The intensity of expression of all the monocyte surface molecules examined was significantly reduced after fish oil supplementation (P < 0.025), although there was no change in the percentage of monocytes expressing each molecule. After incubation with interferon-gamma, there was a similar inhibition of surface molecule expression (with the exception of HLA-DQ) by monocytes from the fish oil-supplemented group, and there was a reduction in the percentage of monocytes expressing both HLA-DR and -DP molecules (P < 0.025). No significant changes were observed in the reference group. Dietary supplementation with fish oil can inhibit the expression of surface molecules involved in the function of human antigen-presenting cells, a potential mechanism by which n-3 fatty acids may suppress cell- mediated immune responses.

How about this:

ORIGINAL RESEARCH COMMUNICATIONS
Fish oil supplementation inhibits the expression of major histocompatibility complex class II molecules and adhesion molecules on human monocytes

DA Hughes, AC Pinder, Z Piper, IT Johnson and EK Lund
Department of Nutrition, Diet and Health, Institute of Food Research, Norwich Laboratory, Norwich Research Park, Colney, Norfolk, United Kingdom.

To test the hypothesis that fish oil supplementation can inhibit the expression of functionally associated molecules on the surface of human blood monocytes, we randomly assigned 12 healthy adults to receive either an n-3 polyunsaturated fatty acid-rich fish oil supplement for 21 d or to receive no supplement. The percentage of monocytes expressing major histocompatibility complex (MHC) class II molecules (HLA-DR, -DP, and -DQ), intercellular adhesion molecule-1, and leukocyte-function-associated antigen-1, and the intensity of expression of each molecule were quantified before and after the study period. Monocytes were examined immediately after blood sampling and again after incubation in serum-free culture medium for 24 h in the presence of interferon-gamma to up-regulate expression of MHC class II molecules by the monocytes. The intensity of expression of all the monocyte surface molecules examined was significantly reduced after fish oil supplementation (P < 0.025), although there was no change in the percentage of monocytes expressing each molecule. After incubation with interferon-gamma, there was a similar inhibition of surface molecule expression (with the exception of HLA-DQ) by monocytes from the fish oil-supplemented group, and there was a reduction in the percentage of monocytes expressing both HLA-DR and -DP molecules (P < 0.025). No significant changes were observed in the reference group. Dietary supplementation with fish oil can inhibit the expression of surface molecules involved in the function of human antigen-presenting cells, a potential mechanism by which n-3 fatty acids may suppress cell- mediated immune responses.

Vary applicable if talking Rhuematoid conditions...of which Busitis is correlated, but not directly related.
From what I've seen (no links or sources to refer to at the moment) n-3 oils are substrate for the production of certain eicasanoids which are less inflammatory than theose produced via arichadonic acid (AA). Both EPA and AA effectively compete for the D-5 or D-6 (not sure which) pathway with a preference for the n-3 acids and therefore a consumption of a larger amount n-3's effectively reduces inflammatory responses within the body. These eicasanoids are actually the same mechanism which NSAIDs inhibit production of, the major difference is that NSAIDs preven production of all eicasanoids, and n-3 supplementation can inhibit or reduce production of specific inflammatory eicasanoids.

Vary applicable if talking Rhuematoid conditions...of which Busitis is correlated, but not directly related.
From what I've seen (no links or sources to refer to at the moment) n-3 oils are substrate for the production of certain eicasanoids which are less inflammatory than theose produced via arichadonic acid (AA). Both EPA and AA effectively compete for the D-5 or D-6 (not sure which) pathway with a preference for the n-3 acids and therefore a consumption of a larger amount n-3's effectively reduces inflammatory responses within the body. These eicasanoids are actually the same mechanism which NSAIDs inhibit production of, the major difference is that NSAIDs preven production of all eicasanoids, and n-3 supplementation can inhibit or reduce production of specific inflammatory eicasanoids.


Thank you for the bit of clarification.

My understanding of this subject is just beginning, but it is very interesting. I have the entire article which explains the process of which they speak. Unfortunately, I don't think it would be looked upon favorably to post a 6 page PDF

Vary applicable if talking Rhuematoid conditions...of which Busitis is correlated, but not directly related.
From what I've seen (no links or sources to refer to at the moment) n-3 oils are substrate for the production of certain eicasanoids which are less inflammatory than theose produced via arichadonic acid (AA). Both EPA and AA effectively compete for the D-5 or D-6 (not sure which) pathway with a preference for the n-3 acids and therefore a consumption of a larger amount n-3's effectively reduces inflammatory responses within the body. These eicasanoids are actually the same mechanism which NSAIDs inhibit production of, the major difference is that NSAIDs preven production of all eicasanoids, and n-3 supplementation can inhibit or reduce production of specific inflammatory eicasanoids.


Thank you for the bit of clarification.

My understanding of this subject is just beginning, but it is very interesting. I have the entire article which explains the process of which they speak. Unfortunately, I don't think it would be looked upon favorably to post a 6 page PDF

Here is something I found

DHAhttp://i136.photobucket.com/albums/q187/vancomycin/dha.jpg

http://i136.photobucket.com/albums/q187/vancomycin/dha2.jpg

Thank you for the bit of clarification.

My understanding of this subject is just beginning, but it is very interesting. I have the entire article which explains the process of which they speak. Unfortunately, I don't think it would be looked upon favorably to post a 6 page PDF

Here is something I found

DHAhttp://i136.photobucket.com/albums/q187/vancomycin/dha.jpg

http://i136.photobucket.com/albums/q187/vancomycin/dha2.jpg

Thanks for the clarification...it is Delta 6 desaturase as the limiting factor. Glad I did not try to say which one for sure, because I would have said the other one....was shooting from memory and would have been wrong. :eek:

My mother uses this for mild arthritis. Supposedly acts like a lube. No idea if it'll help with bursitis though.

http://www.drugstore.com/qxp150364_334918_sespider/nature_made/triple_flex_bone_and_joint_caplets.htm

Thanks for the clarification...it is Delta 6 desaturase as the limiting factor. Glad I did not try to say which one for sure, because I would have said the other one....was shooting from memory and would have been wrong. :eek:


Do you think that it would be prudent to say, that consumption of large amounts of Omega 3's would make less substrates available for the inflammatory pathway of omega 6's, thus reducing the inflammatory response. As well, to say that this response may also blunt the immune response in general, thus reducing systemic inflammation.

Do you think that it would be prudent to say, that consumption of large amounts of Omega 3's would make less substrates available for the inflammatory pathway of omega 6's, thus reducing the inflammatory response. As well, to say that this response may also blunt the immune response in general, thus reducing systemic inflammation.

That is consistent with what I have read. Personally, I am leary of too much n-3's due to the reduction in blood clotting factors...this can be dangerous if carried to too much of an extreme. Since you are working on becoming an RN, I am sure you are quite familiar with how much one can bleed when taking an aspirin or two and then getting cut an hour later. This is due to a lack of the same clotting factors.
Its all about proper balance, but according to some people, the typical American diet is excessive in n-6 FA's and deficient in n-3 FA's leading to a chronic inflammatory situation.

Remember that these "hormones" are how the cells communicate with eachother and have extraordinary immune response modulatory properties.

That is consistent with what I have read. Personally, I am leary of too much n-3's due to the reduction in blood clotting factors...this can be dangerous if carried to too much of an extreme. Since you are working on becoming an RN, I am sure you are quite familiar with how much one can bleed when taking an aspirin or two and then getting cut an hour later. This is due to a lack of the same clotting factors.
Its all about proper balance, but according to some people, the typical American diet is excessive in n-6 FA's and deficient in n-3 FA's leading to a chronic inflammatory situation.

Remember that these "hormones" are how the cells communicate with eachother and have extraordinary immune response modulatory properties.

Ok I am understanding it a bit better now. What we (RN students) are taught is that during times of infection high supplementation of Omega 3's is contraindicated because of the blunting of the immune and inflammatory response.

I was not aware of the anti clotting factors, but if what you say is true there could be a 2 pronged reason for a proper diet that includes more omega 3's and less 6's. As well as reducing endothelial inflammation it "thins" the blood to prevent clotting. Of course one can go the way they always go and get a prescription for plavix and arthrotek, and not have to be concerned with diet.

http://medinfo.ufl.edu/~biochem/Omega.pdf

Part of this at least explains how the eicosanoids made from AA "tend to promote platelet aggregation" and how the eicasanoids made from n-3 fa's tend to exert the opposite effect.
Its 10 pages long in total...if you have the time and patients.