Ah! then this may answer your question... From www.drug.com: "Vitamin K promotes the hepatic formation of active prothrombin (factor II), proconvertin (factor VII), plasma thromboplastin component or Christmas factor (factor IX), and Stuart factor (factor X) {01} {02} {04} {05} {06} , which are required for normal blood clotting. Vitamin K is an essential cofactor for a hepatic microsomal enzyme that catalyzes the post-translational carboxylation of multiple, specific, peptide-bound glutamic acid residues in inactive hepatic precursor proteins of factors II, VII, IX, and X. The resulting gamma-carboxyglutamic acid residues convert the precursor proteins to active coagulation factors that subsequently are secreted by liver cells into the blood. {01}

In healthy individuals, supplemental vitamin K is virtually devoid of pharmacodynamic activity. However, in the presence of vitamin K deficiency, or in the presence of coumarin- or indanedione-derivative anticoagulants, the pharmacologic activity of vitamin K is related to its normal physiological function, which is to promote the hepatic formation of vitamin K-dependent clotting factors {01} .

Vitamin K does not return abnormal platelet function to normal. Vitamin K does not counteract the anticoagulant activity of heparin. "

In other words, don't stress the envelope of the protoplasm enough to cause leakage in the afore mentioned areas that does not have the any medical facilities.