Originally Posted by
Iride01
I think you've missed the point. Certainly burning fat at a higher rate will produce more energy for the muscles to use. However the muscles don't know or care what the source is. And the higher fat burning rate is not going to be enough to produce the energy muscles have the potential to use.. Carbohydrate consumption during that time to maintain serum glucose will still provide energy for the muscles to draw energy from when in zone 2, 3, 4 or 5. And with that carbohydrate they can better meet their full potential since they'll have more available energy.
Even the professional athletes using ketones are not using exclusively ketones. And perhaps it's your suggestion that we buy both. But it seems a even more marginal gain than carb's.
So why is it that riders are ingesting greater and greater amounts of carbs? 120g per hour.. and they would take more.. as much as they can tolerate given GI distress.. correct? What if you can add as much carbs is is tollerable AND... add IN PARALLELL add... IN ADDITION... EVEN MORE energy substrate, that doesn't reduce the amount of carbs you can pack in and doesn't cause increased GI distress as adding even more carbs would? There is a finite energy limit to packing in carbs given GI distress. Exogenous ketones allows riders to effectively break trough that limit by packing in more energy per hr.. ON TOP of carbs that you can max out on.. do you get it now? It's not an either, or... it's a plus, and!
Originally Posted by
Iride01
As for your 5%, that needs some context behind it.
Link to study right here... I provided it!
Bicarbonate Unlocks the Ergogenic Action of Ketone Monoester Intake in Endurance Exercise Chiel Poffé et al. Med Sci Sports Exerc. 2021.
The power for the 15min ITT after the 3hrs was 5% higher for Ketone + Bicarbonate group! That’s an EPO level increase in performance!
”Results: KE ingestion transiently elevated blood D-ß-hydroxybutyrate to ~2-3 mM during the initial 2 h of RACE (P < 0.001 vs CON). In KE, blood pH concomitantly dropped from 7.43 to 7.36 whereas bicarbonate decreased from 25.5 to 20.5 mM (both P < 0.001 vs CON). Additional BIC resulted in 0.5 to 0.8 mM higher blood D-ß-hydroxybutyrate during the first half of IMT180' (P < 0.05 vs KE) and increased blood bicarbonate to 31.1 ± 1.8 mM and blood pH to 7.51 ± 0.03 by the end of IMT180' (P < 0.001 vs KE). Mean power output during TT15' was similar between KE, BIC, and CON at ~255 W but was 5% higher in KE + BIC (P = 0.02 vs CON). Time to exhaustion in the sprint was similar between all conditions at ~60 s (P = 0.88). Gastrointestinal symptoms were similar between groups.”